Search results for " infiltration"

showing 10 items of 104 documents

Estimating the macroscopic capillary length from Beerkan infiltration experiments and its impact on saturated soil hydraulic conductivity predictions

2020

International audience; The macroscopic capillary length, λc, is a fundamental soil parameter expressing the relative importance of the capillary over gravity forces during water movement in unsaturated soil. In this investigation, we propose a simple field method for estimating λc using only a single-ring infiltration experiment of the Beerkan type and measurements of initial and saturated soil water contents. We assumed that the intercept of the linear regression fitted to the steady-state portion of the experimental infiltration curve could be used as a reliable predictor of λc. This hypothesis was validated by assessing the proposed calculation approach using both analytical and field d…

010504 meteorology & atmospheric sciencesCapillary actionField dataHydraulic conductivity0207 environmental engineeringSoil science02 engineering and technology[SDV.SA.SDS]Life Sciences [q-bio]/Agricultural sciences/Soil study01 natural sciencesHydraulic conductivityBeerkan Hydraulic conductivity Infiltration Macroscopic capillary length Ring infiltrometerApproximation errorBeerkanLinear regressionSettore AGR/08 - Idraulica Agraria E Sistemazioni Idraulico-Forestali[SDU.STU.HY]Sciences of the Universe [physics]/Earth Sciences/Hydrology020701 environmental engineeringRing infiltrometer0105 earth and related environmental sciencesWater Science and TechnologyInfiltration6. Clean waterMacroscopic capillary lengthInfiltration (hydrology)Capillary lengthSoil waterEnvironmental science
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Laboratory evaluation of falling-head infiltration for saturated soil hydraulic conductivity determination

2020

Falling-head one-dimensional infiltration procedures, such as the simplified falling-head (SFH) technique, yield estimates of saturated soil hydraulic conductivity, Ks, with parsimonious and rapid experiments. Factors that can influence determination of Ks by the SFH technique were tested in the laboratory on three repacked soils differing by particle diameter ranges (0-2000, 0- 105 and 105-2000 mm, respectively). Using the theoretically calculated depth of ponding on the infiltration surface, D, instead of the measured one had a small impact on the Ks calculations (means differing by a factor of 1.1-1.2, depending on the soil). For the finest soil, Ks decreased by 3.1 times as D increased …

010504 meteorology & atmospheric sciencesMechanical Engineeringlcsh:SBioengineering04 agricultural and veterinary scienceslcsh:S1-97201 natural sciencesIndustrial and Manufacturing Engineeringlcsh:AgricultureInfiltration (hydrology)Saturated soil hydraulic conductivityHydraulic conductivityLaboratory investigationFalling-head one-dimensional infiltrationSimplified falling-head techniquesimplified falling-head technique.040103 agronomy & agricultureSettore AGR/08 - Idraulica Agraria E Sistemazioni Idraulico-Forestali0401 agriculture forestry and fisheriesEnvironmental scienceGeotechnical engineeringlcsh:Agriculture (General)0105 earth and related environmental sciences
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Antagonization of IL-17A Attenuates Skin Inflammation and Vascular Dysfunction in Mouse Models of Psoriasis

2019

Besides skin inflammation, patients with severe psoriasis suffer from an increased risk of cardiovascular mortality. IL-17A plays a central role in the development of psoriasis and might connect skin and vascular disease. The aim of this study was to clarify whether anti-IL-17A therapy could also ameliorate the vascular dysfunction associated with severe psoriasis. We analyzed three murine models with varying severities of psoriasis-like skin disease concerning their vascular function and inflammation: (i) K14-IL-17A(ind/+) mice with keratinocyte-specific IL-17A overexpression and an early-onset severe psoriasis-like phenotype; (ii) homozygous CD11c-IL-17A(ind/ind) and heterozygous CD11c-IL…

0301 basic medicineInflammationDermatologyDiseasemedicine.disease_causeBiochemistryProinflammatory cytokine03 medical and health sciencesMice0302 clinical medicinePsoriasis Area and Severity IndexPsoriasismedicineAnimalsHumansPsoriasisVascular DiseasesAntibodies BlockingMolecular BiologyAortaSkinInflammationImiquimodintegumentary systembusiness.industryVascular diseaseInterleukin-17Cell Biologymedicine.diseaseAngiotensin II3. Good healthMice Inbred C57BLDisease Models AnimalOxidative Stress030104 developmental biologyNeutrophil Infiltration030220 oncology & carcinogenesisImmunologyDisease Progression[SDV.IMM]Life Sciences [q-bio]/ImmunologyImmunotherapymedicine.symptombusinessReactive Oxygen SpeciesOxidative stress
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The Impact of Small Extracellular Vesicles on Lymphoblast Trafficking across the Blood-Cerebrospinal Fluid Barrier In Vitro.

2020

Central nervous System (CNS) disease in pediatric acute lymphoblastic leukemia (ALL) is a major concern, but still, cellular mechanisms of CNS infiltration are elusive. The choroid plexus (CP) is a potential entry site, and, to some extent, invasion resembles CNS homing of lymphocytes during healthy state. Given exosomes may precondition target tissue, the present work aims to investigate if leukemia-derived exosomes contribute to a permissive phenotype of the blood-cerebrospinal fluid barrier (BCSFB). Leukemia-derived exosomes were isolated by ultracentrifugation from the cell lines SD-1, Nalm-6, and P12-Ichikawa (P12). Adhesion and uptake to CP epithelial cells and the significance on sub…

0301 basic medicineIntegrinexosomesEndocytosisExosomecentral nervous system infiltrationCatalysisArticlepediatric acute lymphoblastic leukemiaInorganic Chemistrylcsh:Chemistry03 medical and health sciencesExtracellular Vesicles0302 clinical medicineCell MovementCentral Nervous System DiseasesCell Line TumorCell AdhesionAnimalsHumansNeoplasm InvasivenessLymphocytesPhysical and Theoretical ChemistryMolecular Biologylcsh:QH301-705.5Spectroscopychoroid plexusbiologyChemistryLymphoblastOrganic ChemistryEpithelial CellsGeneral MedicinePrecursor Cell Lymphoblastic Leukemia-LymphomaHematopoietic Stem CellsMicrovesiclesEndocytosisComputer Science ApplicationsCell biologyProtein Transport030104 developmental biologylcsh:Biology (General)lcsh:QD1-999Cell cultureBlood-Brain Barrier030220 oncology & carcinogenesisbiology.proteinChoroid plexusHoming (hematopoietic)International journal of molecular sciences
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Loss of cellular FLICE-inhibitory protein promotes acute cholestatic liver injury and inflammation from bile duct ligation.

2017

Cholestatic liver injury results from impaired bile flow or metabolism and promotes hepatic inflammation and fibrogenesis. Toxic bile acids that accumulate in cholestasis induce apoptosis and contribute to early cholestatic liver injury, which is amplified by accompanying inflammation. The aim of the current study was to evaluate the role of the antiapoptotic caspase 8-homolog cellular FLICE-inhibitory (cFLIP) protein during acute cholestatic liver injury. Transgenic mice exhibiting hepatocyte-specific deletion of cFLIP (cFLIP−/−) were used for in vivo and in vitro analysis of cholestatic liver injury using bile duct ligation (BDL) and the addition of bile acids ex vivo. Loss of cFLIP in h…

0301 basic medicineLiver CirrhosisTime FactorsPhysiologyCASP8 and FADD-Like Apoptosis Regulating ProteinInflammationApoptosisp38 Mitogen-Activated Protein KinasesHepatitisBile Acids and Salts03 medical and health sciencesNecrosisCholestasisPhysiology (medical)medicineHepatic Stellate CellsAnimalsASK1Genetic Predisposition to DiseaseLigationCells CulturedTumor Necrosis Factor alpha-Induced Protein 3chemistry.chemical_classificationLiver injuryCommon Bile DuctMice KnockoutReactive oxygen speciesHepatologyBile duct ligationGastroenterologyTranscription Factor RelAmedicine.diseaseOxidative Stress030104 developmental biologyCholedocholithiasisPhenotypechemistryLiverNeutrophil InfiltrationApoptosisFLICE Inhibitory ProteinCancer researchHepatocytesCytokinesmedicine.symptomInflammation MediatorsSignal TransductionAmerican journal of physiology. Gastrointestinal and liver physiology
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Intratumoral immunosuppression profiles in 11q-deleted neuroblastomas provide new potential therapeutic targets

2021

In this issue, Coronado et al. attempt to improve our understanding of the factors affecting the response to immunotherapy in a large subset of high‐risk neuroblastoma with hemizygous deletion of chromosome 11q. By using several computational approaches, the authors study potential transcriptional and post‐transcriptional pathways that may affect the response to immunotherapy and further be leveraged therapeutically in a biomarker‐directed fashion.

0301 basic medicineMaleCancer Researchmedicine.medical_treatmentRetinoic acidchemistry.chemical_compoundNeuroblastoma0302 clinical medicineTumor Microenvironment11q deletion anti-GD2 immunotherapy combination immunotherapy immune cell infiltration miRNAs neuroblastomaMedicineeducation.field_of_studyimmune cell infiltration11q deletionImmunosuppressionGeneral Medicinelcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensPatologiaNeoplasm ProteinsmicroRNAsSurvival Rateanti‐GD2 therapyOncology030220 oncology & carcinogenesiscombination immunotherapymiRNAsMolecular Medicineimmune checkpoint inhibitionFemaleImmunotherapyChromosome Deletionanti‐GD2 immunotherapyPopulationlcsh:RC254-282Disease-Free Survival03 medical and health sciencesImmune systemNeuroblastomaGeneticsImmune ToleranceHumanseducationRetrospective Studiesbusiness.industryChromosomes Human Pair 11Immunotherapymedicine.diseaseImmune checkpointBlockade030104 developmental biologychemistryCancer researchCommentarybusiness
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Gut Microbiota Restricts NETosis in Acute Mesenteric Ischemia-Reperfusion Injury.

2020

Objective: Recruitment of neutrophils and formation of neutrophil extracellular traps (NETs) contribute to lethality in acute mesenteric infarction. To study the impact of the gut microbiota in acute mesenteric infarction, we used gnotobiotic mouse models to investigate whether gut commensals prime the reactivity of neutrophils towards formation of neutrophil extracellular traps (NETosis). Approach and Results: We applied a mesenteric ischemia-reperfusion (I/R) injury model to germ-free (GF) and colonized C57BL/6J mice. By intravital imaging, we quantified leukocyte adherence and NET formation in I/R-injured mesenteric venules. Colonization with gut microbiota or monocolonization with Esch…

0301 basic medicineMaleExtracellular TrapsMesenteric infarctionLipopolysaccharideNeutrophilsGut floraExtracellular Traps03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAcute mesenteric ischemiaVenulesmedicineCell AdhesionEscherichia coliLeukocytesAnimalsGerm-Free LifeLeukocyte RollingMesenteryCells CulturedMice Knockoutbiologybusiness.industryNeutrophil extracellular trapsbiology.organism_classificationmedicine.diseaseGastrointestinal MicrobiomeMice Inbred C57BLToll-Like Receptor 4Disease Models Animal030104 developmental biologychemistryNeutrophil Infiltration030220 oncology & carcinogenesisMesenteric IschemiaReperfusion InjuryImmunologyHost-Pathogen InteractionsFemaleCardiology and Cardiovascular MedicinebusinessReperfusion injuryBacillus subtilisSignal TransductionArteriosclerosis, thrombosis, and vascular biology
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PD-L1 Expression and Immune Cell Infiltration in Gastroenteropancreatic (GEP) and Non-GEP Neuroendocrine Neoplasms With High Proliferative Activity

2019

The potential of neuroendocrine neoplasms (NEN) to respond to checkpoint inhibitors is largely unknown and full of great expectations. Immunohistochemical (IHC) studies of programmed cell death ligand 1 (PD-L1) expression in the tumor microenvironment and its implications in predicting the response to checkpoint inhibition is a very active subject. Currently, the combined analysis of PD-L1 expression and tumor-associated immune cell (TAIC) infiltration is considered the best predictive marker of therapeutic response. Here we investigated the expression of PD-L1 on tumor cells (TC) and tumor-infiltrating immune cells (IC) by IHC in 68 NEN samples with a high proliferation rate (Ki-67 >20%…

0301 basic medicinePD-L1Cancer ResearchCD3immune checkpoint inhibitorBiologyNeuroendocrine tumorslcsh:RC254-28203 medical and health sciences0302 clinical medicineImmune systemPD-L1tumor associated immune cellmedicineT cell infiltrationOriginal ResearchTumor microenvironmentneuroendocrine neoplasmCD68neuroendocrine carcinomamedicine.diseaselcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogens030104 developmental biologyOncology030220 oncology & carcinogenesisCancer researchbiology.proteinImmunohistochemistryCD8neuroendocrine tumorFrontiers in Oncology
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Unusual presentation of blastic plasmacytoid dendritic cell neoplasm: Pitfalls in other hematolymphoid neoplasms

2020

Abstract Blastic plasmacytoid dendritic cell neoplasm (BPDCN) is a rare CD4+/CD56+ hematological malignancy with aggressive clinical course and poor prognosis. Histologically, BPDCN is characterized by a diffuse, monomorphous infiltration of cutaneous, subcutaneous, and sometimes other tissues such as lymph nodes and bone marrow, by medium-sized neoplastic cells with blastoid morphology. Typically, there is absence of lymphocytic infiltrate. Diagnosis relies on immunophenotypic expression of CD4, CD56, and the more specific markers of plasmacytoid dendritic cells CD123, CD303/BDCA2, and TCL1. We report a case of a 57-year-old man who presented a 4 cm-long solitary, erythemateous lesion on t…

0301 basic medicinePathologymedicine.medical_specialtyMyeloidMYCASXL1BlastoidPathology and Forensic MedicineLymphocytic Infiltrate03 medical and health sciences0302 clinical medicineBlastic plasmacytoid dendritic cell neoplasmT-cells infiltrationlcsh:PathologyMedicineTET2biologybusiness.industryETV6medicine.diseasebiology.organism_classificationLymphomaETV6030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisInterleukin-3 receptorBone marrowbusinessInfiltration (medical)lcsh:RB1-214Human Pathology: Case Reports
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AB0458 A PHASE II RANDOMISED, DOUBLE-BLIND, PLACEBO-CONTROLLED, PROOF OF CONCEPT STUDY OF ORAL SELETALISIB IN PATIENTS WITH PRIMARY SJÖGREN’S SYNDROM…

2019

Background: Seletalisib is a potent, selective oral inhibitor of phosphoinositide-3 kinase delta (PI3Kδ). Preclinical data have shown that the PI3Kδ pathway is upregulated within salivary glands of patients with PSS and contributes to disease pathogenesis.1 Objectives: To assess the efficacy and safety of seletalisib in patients with PSS. Methods: In this Phase II, double-blind, proof of concept study (NCT02610543), patients with PSS having an EULAR Sjogren’s Syndrome Disease Activity Index (ESSDAI) score ≥5 were randomised 1:1 to seletalisib once daily or placebo (PBO) in addition to current PSS therapy for 12 weeks. The primary endpoint was change from baseline in ESSDAI at Week 12. The s…

030203 arthritis & rheumatology0301 basic medicinemedicine.medical_specialtyLymphocytic infiltrationbusiness.industryPlaceboPreclinical dataDouble blind03 medical and health sciences030104 developmental biology0302 clinical medicineTolerabilitySeletalisibInternal medicinemedicineIn patientSjogren sbusiness
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